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  1. #651
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    Quote Originally Posted by piwanoi View Post
    Hmm , I'm a bit of a newcomer to this forum ,but hardly a newcomer to Thailand ,I have read the thread from start to finish of all the "theory's" etc ,but on reading Hypatia's post#614 ,sadly I have to nod my head in full agreement to it, because in a nutshell it just about says it all (IMHO) !.
    Gosh, thanks, What did I say?
    Oh dehydration, yes after vomiting..

    GiT I agree many many many are being poisoned, I'm not sure it's always meant to kill, but as a say let's give them diarrhea for complaining about lunch.
    My point is each will have different symptoms, and dehydration is a major factor. Vomiting causes dehydration, leads to organ failure and the state of health is also a factor in how the body begins to shut down.
    So to look for one set of symptoms and align it with one poison is fruitless.
    Probably all kinds of poisons being used.
    What's important is that it is happening, a lot and still we are being told it is not connected or relevant.

  2. #652
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    Quote Originally Posted by Hypatia View Post
    Quote Originally Posted by alitongkat View Post
    it looks like poison, it looks like fish poisoning, but it isnt fish poisoning...

    what could it be?

    THEY MUST have tested for methomyl...

    therefore, it should be ruled out from the possible sources...
    My Dear Ali...
    Pleases remember samples just weren't taken soon enough and in a manner consistent with competent investigations, on any victims, as of yet. The actual compound and mechanism is a mystery, and well not to be rude to you , but you are posting to yourself now.
    We all have our theory, seems we have to wait for another death and then maybe that one will get proper pathology tests.
    the young western female demo is so skewed, I do not see ow anyone can think it is accidental.
    its not true... some of the cases must have been extensively tested, as they spent hours in hospital before they died/survived...

    phi phi: 1 girl survived (treated, 20something), one died at arrival in the hospital... (scandinavian and american)

    phuket(?): arabian girl (20something), flue like symptoms, hospital, released, dead next morning

    cambodia: 2 girls (end 20s), both in hospital, one died in hospital, the other one too, 2 days later or so...

    chiang mai: at least two girls (20s) in hospital, one died there

    plenty of tests and immediate autopsy possible...

    as ALL of them died of "unknown" causes in a local hospital, it CANT have been methomyl...
    Last edited by alitongkat; 26-11-2012 at 10:16 PM.

  3. #653
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    more "cures" ? (for what i understand...)

    Lemon balm - enhances cholinesterase receptors

    "white flood" or similar (nitric oxide)
    hese results show that inhibition of NOS, as well as neuronal NOS, significantly reduces AChE inhibitor-induced muscle cell degeneration, suggesting that increased nitric oxide production mediates such myopathy.

  4. #654
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    Quote Originally Posted by superman View Post
    Atropine is not an actual antidote for organophosphate poisoning . However, by blocking the action of acetylcholine at muscarinic receptors, atropine also serves as a treatment for poisoning by organophosphate insecticides and nerve gases.
    "An antidote is a substance which can counteract a form of poisoning".[1]
    Antidote - Wikipedia, the free encyclopedia

    Atropine is an antidote to both types of poisoning.

    Note emedicine's use of the word 'antidote'

    "Antidotes

    Class Summary

    Anticholinergics, such as atropine, cause pharmacologic antagonism of excess anticholinesterase activity at muscarinic receptors".
    Last edited by guyinthailand; 27-11-2012 at 03:50 AM.

  5. #655
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    Quote Originally Posted by alitongkat View Post
    , its NOT recommended to give activated charcoal, i


    If the patient has no breathing difficulties and has recently swallowed the poison and you are far from a hospital, then activated charcoal is absolutely recommended.


    the risk you run in having them eat an entire bottle (100 caps, minimum, for a large ingestion) is that they could start to vomit and aspirate the stuff--dangerous if you're not near a hospital where patient can be intubated to support breathing. But you have to 'risk' it if you can't get patient to hospital soon.

    It is ideal to go to a hospital ASAP and let them give the charcoal there while being prepared to intubate.

    Charcoal can easily save the life of someone recently poisoned with just about any poison EXCEPT:
    Activated carbon does not bind well to certain chemicals, including alcohols, glycols, strong acids and bases, metals and most inorganics, such as lithium, sodium, iron, lead, arsenic, fluorine, and boric acid.

  6. #656
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    Quote Originally Posted by alitongkat View Post
    treatment:

    apart from atropine, its "oxime" (pralidoxime) to treat severe carbamite (Methomyl) poisoning...

    ...when a poisoned patient presents with a cholinergic crisis and the poison is unknown, or an exposure to a mixture of cholinesterase-inhibitors (organophospate and carbamate) has occurred, treatment with a combination of atropine and oximes is advised...

    advised to use atropine in conjunction with oxime therapy even when carbamate intoxication is present with high certainty (with no information regarding the specific type of carbamate).

    According to the textbook Medical Toxicology by Dart (2003)18, in many cases of carbamate exposure pralidoxime has been shown to be either beneficial, or at least not harmful.
    http://igitur-archive.library.uu.nl/...254_Thesis.pdf

    Some authors advise caution in using pralidoxime for carbamate poisoning because, unlike organophosphate poisoning, the bond made between the carbamate and cholinesterase is reversible by itself and so by giving the pralidoxime (which also reverses it) you can end up causing too much cholinesterase to be there. You can use it in organophosphate poisoning, too, but only if given within 30 minutes of poisoning because any later and the bond has become irreversible--it has 'aged'). But, yes, if patient presents soon and you aren't sure if he has been poisoned by carbamates or organophosphates, or both, use of pralidoxime might be warranted. However, the ANTIDOTE atropine works in both cases.

    and emedicine says:
    "(praalidoxime is) Rarely needed in carbamate poisonings"
    Medscape: Medscape Access

    Note emedicine's use of the word 'antidote'

    "Antidotes

    Class Summary

    Anticholinergics, such as atropine, cause pharmacologic antagonism of excess anticholinesterase activity at muscarinic receptors".
    Last edited by guyinthailand; 26-11-2012 at 11:18 PM.

  7. #657
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    Quote Originally Posted by alitongkat View Post

    so we have to assume, they were tested and no carbamite was found...
    I mentioned earlier that carbamates are hard to detect post mortem. For the test, marker cholinesterase is used and since carbamate-cholinesterase bond is reversible all by itself, by the time they test it, everything seems back to normal.

    But maybe they have some new techniques now.

  8. #658
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    Quote Originally Posted by alitongkat View Post

    its not true... some of the cases must have been extensively tested, as they spent hours in hospital before they died/survived...

    phi phi: 1 girl survived (treated, 20something), one died at arrival in the hospital... (scandinavian and american)

    phuket(?): arabian girl (20something), flue like symptoms, hospital, released, dead next morning

    cambodia: 2 girls (end 20s), both in hospital, one died in hospital, the other one too, 2 days later or so...

    chiang mai: at least two girls (20s) in hospital, one died there

    plenty of tests and immediate autopsy possible...

    as ALL of them died of "unknown" causes in a local hospital, it CANT have been methomyl...
    Quote Originally Posted by guyinthailand View Post
    Quote Originally Posted by alitongkat View Post

    so we have to assume, they were tested and no carbamite was found...
    I mentioned earlier that carbamates are hard to detect post mortem. For the test, marker cholinesterase is used and since carbamate-cholinesterase bond is reversible all by itself, by the time they test it, everything seems back to normal.

    But maybe they have some new techniques now.
    Ok.... the debate has devolved into an interesting point. Let's try to find out the reality of it, shall we ? Perhaps I should phone a poisons information centre here in Australia ?
    .
    .
    .
    .

  9. #659
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    carbamates ARE hard to detect...

    its because its a "reversible" thing, that means, in the first hour there is a high "value", and a few hours later its "reversed" (back to normal)...

    but nevertheless, you will die of it...

    so this means, it DOES DO something (more)...
    its not only about diminishing the liver enzyme, but it has destructive effects (on cells...)

    so this destruction must be able to see, even though the enzyme is back to almost normal values... (normal is btw dependend on the individual, some have higher normals, others lower...)...

  10. #660
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    the NL study also said, it can be detected in the hair...

    but dont know how long it takes to make it there?
    does it go within hours to the root?


    just saw this on wiki, its some more explanation...
    The so-called carbamate insecticides feature the carbamate ester functional group.

    Included in this group are
    -- aldicarb, carbofuran (Furadan), carbaryl (Sevin), ethienocarb, fenobucarb, oxamyl and methomyl.
    These insecticides kill insects by reversibly inactivating the enzyme acetylcholinesterase.

    -- The organophosphate pesticides also inhibit this enzyme, although irreversibly, and cause a more severe form of cholinergic poisoning.[2]
    http://en.wikipedia.org/wiki/Carbamate


    (( "Irreversibly" means (faik), that it might take weeks to reverse... ))
    Last edited by alitongkat; 27-11-2012 at 09:01 AM.

  11. #661
    Thailand Expat superman's Avatar
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    Quote Originally Posted by guyinthailand
    Quote: Originally Posted by superman View Post Atropine is not an actual antidote for organophosphate poisoning . However, by blocking the action of acetylcholine at muscarinic receptors, atropine also serves as a treatment for poisoning by organophosphate insecticides and nerve gases. "An antidote is a substance which can counteract a form of poisoning".[1] Antidote - Wikipedia, the free encyclopedia Atropine is an antidote to both types of poisoning.
    Atropine is not an actual antidote for organophosphate poisoning.
    Atropine - Wikipedia, the free encyclopedia

  12. #662
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    Deaths Of Quebec Women In Thailand May Have Been Caused By Pesticide
    03/13/2014

    A highly toxic pesticide used to control bedbugs in some holiday hotels in Asia may have caused the mysterious deaths of two Quebec sisters travelling in Thailand as well as several other tourists, according to new evidence from a joint investigation by CBC’s the fifth estate and Radio-Canada’s Enquete.

    Audrey and Noémi Bélanger set off on a trip through Thailand in 2012. Days after they arrived at the popular tourist destination of Phi Phi Island, a maid found the sisters dead in their hotel room. Both were covered in vomit, with their fingernails and toenails tinged blue.

    “I let out a scream for my older girl and I let out a scream for my youngest. I didn’t know what else to do,” said their mother, Linda Bélanger, upon hearing the news.

    At the time, local authorities suggested several possible causes, from food poisoning to drugs. The Bélangers requested that the Quebec coroner do an autopsy on their daughters. Almost two years since the autopsy was performed, the results have not been released. They are expected to be made public in June.

    The CBC/Radio-Canada investigation received a tip about what may have killed the sisters that points to a lethal pesticide called aluminum phosphide.

    In Canada, the use of this pesticide is strictly regulated, and fumigators must get six months of training before they can handle it. Denis Bureau, a fumigation specialist in Quebec who is licensed to use it, told the fifth estate’s Mark Kelley that it could kill a human in less than two hours if the concentration is high enough.

    “If you’re asleep in the room next to it or in the room where it’s been under fumigation, you’ll be dead in a few hours,” he said.

    Bureau said aluminum phosphide is commonly used to kill insects in large spaces such as grain silos or ship cargo holds. In Canada, it is not permitted to be used in spaces like homes or hotels. It is typically found in the form of pellets that, once exposed to air and moisture, release a poisonous gas called phosphine.

    In Thailand, authorities told the CBC/Radio-Canada investigation that the use of aluminum phosphide in hotels is strictly forbidden.

    A team from ​Enquête went undercover to visit seven pest control companies in Thailand to see if it is possible to get the pesticide for use in a hotel.

    Employees at most of the companies said they did not use this pesticide in hotels. But at one company, the owner explained how simple it is to use aluminum phosphide pellets to kill bedbugs in a hotel or guest house. After three days, she said a room fumigated with aluminum phosphide would be safe to sleep in.

    Not the only victims

    The CBC/Radio-Canada investigation learned that the Bélanger sisters are not the only travellers whose deaths may be linked to this pesticide. In May of 2009, two other tourists staying on Phi Phi Island also died mysteriously.​

    Norwegian Julia Bergheim and American Jill St. Onge were staying in adjacent rooms at the Laleena Guest House, and they experienced similar symptoms including vomiting, dizziness and blue fingernails and toenails. Both were dead within 24 hours.

    Four years after her daughter’s death, Bergheim’s mother, Ina Thoresen, received a report from the Norwegian Institute of Public Health. Authorities there had consulted with leading experts from around the world about what happened to her daughter. Though they could not state the exact cause of Bergheim’s death, they concluded that the most likely cause was poisoning from the phosphine gas released by the pesticide.

    The Norwegian report also states that Canadian medical examiners found traces of the gas from aluminum phosphide in the bodies of the Bélanger sisters.

    Toxicologist Joel Mayer said that the symptoms experienced by the Bélanger sisters, Bergheim and St. Onge are typical of someone exposed to aluminum phosphide.

    Mayer said blue fingernails and toenails are a classic sign of oxygen deprivation.

    “It’s a respiratory poison, affecting the transport of oxygen … or interfering with the utilization of oxygen by various cells in the body,” he told Kelley.

    In all, since 2009, there have been about a dozen suspicious deaths of other tourists in Thailand, some of which may be linked to the misuse of pesticides.

    In 2011, four people staying in a hotel in the northern Thai city of Chiang Mai died within a few days. After those deaths, a Thai investigation suggested pesticides might have been the cause, and recommended further study of the risk.

    'One of the emerging problems'

    At Thailand’s Ministry of Health, Dr. Pasakron Akarasew said there is growing concern about pesticides.

    “Chemical poisoning is one of the emerging problems. So we hope we can do the job better in the future,” he said.

    He seemed surprised to hear that aluminum phosphide might be used in hotel rooms, since it is dangerous and illegal.

    Still, he said Thailand is as safe as any other country.

    “I may travel someplace and get the same problem,” he said. “I may go to Europe, Canada, Australia and can get the same problem like this.”

    But Thoresen, Julia Bergheim’s mother, does not believe that Thailand is a safe place to visit.

    “It’s a calculated risk to go to Thailand. You think that it’s safe to go there, but it’s not,” she said. “If anything happens, then you can’t get the help that you will get at home. Not from the police, not from the authorities, maybe not in the hospitals. I don’t know, that’s my experience.”

    Last October, Carl and Linda Bélanger got a letter of condolence from the Thai Ministry of Tourism and Sports. In it, they were offered the equivalent of about $20,000 US in insurance coverage to compensate for their loss.

    The Bélangers refused the money, and told Kelley the letter will not stop them from speaking out about what happened to their daughters. They want to put pressure on Thai authorities to prevent more deaths.

    “If it was really a pesticide that was put in the bedroom and it was that powerful, it was negligence,” said Linda Bélanger. “I don’t want there to be any more people who die due to negligence.”

    huffingtonpost.ca

  13. #663
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    Quote Originally Posted by Mid
    In Thailand, authorities told the CBC/Radio-Canada investigation that the use of aluminum phosphide in hotels is strictly forbidden.
    a small IR driven chromatograph that you can plug into the microUSB port on your phone may be an essential travel item

    iPhone turns into a spectrometer - News - spectroscopyNOW.com

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    I said in August 2011 it was aluminium phosphide in a thread on TD about the Downtown Inn deaths :
    Quote Originally Posted by Latindancer View Post

    I have another hypothesis, which I think fits very well. It's very compelling, in fact.....Aluminium Phosphide poisoning.
    Keeping in mind that the panel of experts has stated that it is likely a poison of some unknown sort, consider the following, and please note that both metabolic acidosis and myocarditis are present :
    Aluminium phosphide poisoning.

    And the following small PDF file (from Thailand):
    http://164.115.5.58/seminar/file/201...bilitymode.pdf

    Aluminium Phosphide is readily available in Thailand and used as a rat poison or grain fumigant. Only 1.5 grams will kill a person. It is quite possible that some dust from pellets of this became mixed with grain eaten by the deceased in the Downtown Inn.

    Just Google :
    'aluminium phosphide' poisoning acidosis myocarditis
    and look at a few of the results. It's the commonest lethal cause of poisonings (and suicides) in developing countries.
    It's readily available here. It would have been easy for it to have gotten into food there, and the symptoms fit.
    There is one prominent symptom which it would be interesting to ask the two survivors if they had : a decaying fish, or garlic-like smell.
    One of them had very low blood pressure, which is also a symptom of Al Phosphide poisoning.
    I just looked at the website her father created. http://www.thailandtraveltragedies.c...carter-tragedy
    https://teakdoor.com/thailand-and-asi...-death-24.html (NZ woman in Thailand food poisoning death)

    Inappropriate use of pesticides happens often in Thailand

    Quote Originally Posted by Latindancer View Post
    I lived in Chiang Mai for 6 months in 1998 and read in a local paper then about a poor woman who had been employed working on a vegetable farm somewhere in the North, and who had brought home some veggies. She fed some to her 8 year old daughter who then died. I felt very sad about this, as the woman was quite distraught.
    The stupid part was that her employer had been using a pesticide on vegetables, that was only meant to be used on FLOWERS !
    In the same article they also showed actual photos of people elsewhere applying pesticide to crops with their bare hands.
    Last edited by Latindancer; 15-03-2014 at 01:44 PM.

  15. #665
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    Give yourself a Blue Peter badge mate, pesticide was identified as the killer ages ago.

    They even raided the pest control company that did the spraying, so they knew as well.

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    Ah, but they were talking about chlorpyrifos ! People don't spray aluminium phosphide.

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    Like it matters now.

  18. #668
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    Looks like a few TD posters may have got it right:

    Still not 100% certain but it looks as good as it's going to get.

    The Globe and Mail March 2, 2015

    The same type of pesticide that killed two children in Fort McMurray last month has been identified by a coroner as the possible culprit in the death of two Quebec sisters who were found unconscious in their Thailand hotel room in 2012.

    In a report released Monday, the coroner Renée Roussel said phosphine, the gas released by aluminum phosphide, is the most likely poison that killed killed Audrey Bélanger, 20, and her sister Noémi, 25, while they were staying on Phi Phi Islands.

    The two sisters from Pohénégamook, Que., were part of a cluster of travellers who died in unexplained circumstances in Southeast Asia.

    Dr. Roussel said she was unable to formally confirm phosphine's role because it left no traces in the remains of the Bélanger sisters. Lengthy toxicology tests failed to find signs of nearly 800 possible poisonous chemicals, Dr. Roussel told reporters as she released her report Monday.

    The only substances the toxicology tests detected were proguanil, a medication to prevent malaria, and N,N-diethyl-m-toluamide, the insect repellent widely known as Deet.

    The Thai autopsy initially blamed Deet as the cause of death. However, the Deet levels found in the sisters’ blood was too low to be toxic and were consistent with the substance being applied in normal doses on the skin, Dr. Roussel said.

    The investigation then looked at substances that could kill quickly but would be so volatile they wouldn’t leave traces. Dr. Roussel said this pointed at aluminum phosphide. A dangerous but cheap farming pesticide, aluminum phosphide is widely used in developing countries.

    When exposed to moisture in the air, aluminum phosphide releases a toxic gas, phosphine. Last month, a two-year-old boy, Zia Hassan, and his eight-month-old sister, Zara, died after their parents used phosphine brought into Canada from Pakistan to try to fumigate their Fort McMurray, Alta., apartment.

    Following the two children's death, Health Canada said phosphine pesticides can only be sold to licensed professionals and are not approved for use against bedbugs.
    Similarly, in Thailand, it is illegal to use phosphine indoors, but Dr. Roussel said “we believe it was likely used anyway.”

    Her report noted that the region's innkeepers struggle to contain insects, especially bedbugs in hotel rooms. Brain-cell samples from the sisters showed signs of trauma caused by an acute lack of oxygen, a symptom consistent with exposure to aluminum phosphide, the coroner said.

    Her report noted that, since 2009, about 20 foreign travellers have died in similar fashion in southeast Asia. Two women, Julie Bergheim of Norway and Jill St. Onge of the United States, died in 2009 at another hotel in Phi Phi, with symptoms similar to the Bélanger sisters.

    The report said FBI agents consulted Canadian investigators and came to similar conclusions pointing at aluminum phosphide.

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    It seems that aluminum phosphide poisoning was the cause of death in the unexplained fatalities in this case and in Chiang Mai.

    The use of this pesticide is prohibited in public places here in Thailand but then, that means nothing in a country which has no rule of law, a dysfunctional judiciary and no effective enforcement of any legislation of any sort.

    Travellers should be made aware by their embassies that accommodation in the lower end of the market here could prove fatal given the reckless stupidity of their owners.

    Certainly, any attempt to gain compensation through litigation here would be as futile as Quasimodo getting a freebie from Dirty Noi because he had a hunch she was nice.

  20. #670
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    Quote Originally Posted by baldrick View Post
    Quote Originally Posted by Mid
    In Thailand, authorities told the CBC/Radio-Canada investigation that the use of aluminum phosphide in hotels is strictly forbidden.
    a small IR driven chromatograph that you can plug into the microUSB port on your phone may be an essential travel item

    iPhone turns into a spectrometer - News - spectroscopyNOW.com
    Excellent, can add that to the amateur sleuth kit and get the CSI:TD Junior Detective badge!

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    Thais admit it was 'a' pesticide

    I recently came across this and related poisoning-deaths-in-Thailand threads and was fascinated with them and read them all. Well, well…finally, years after the deaths, a ‘Thai authority’ finally admits, on camera, in 2014, it was ‘a pesticide’ but only because toxicologists in Norway and Canada ‘forced’ them to admit what some on Teakdoor figured out in 2011. I was not surprised to find the Thais making every excuse under the sun for the deaths, 6 in Chiang Mai and 4 in Koh Phi Phi. The Thais blamed ‘coincidence’, food and fish, allergies, mushrooms, drugs, DEET (supposedly consumed by deceased in ‘bucket drinks’). And after listening to the Thai authorities say aluminum phosphide is strictly regulated, you gotta love the undercover work of CBC Canada taping the Thai pesticide woman saying “Sure, I can put some aluminum phosphide in whatever rooms you want”. Here, it’s all ‘on tape’, including a clip of the journalists swabbing the rooms to have the samples analyzed.

    video:
    http://www.cbc.ca/player/play/2441757444/

    The same team does another, more in-depth, look at the deaths with a focus on how the Thai authorities handled it. The scene where the journalists are asking the uniformed Thai official about the report sitting on his desk regarding the deaths, one journalist points to the report on his desk with Thai writing on the cover and asks him, “What does it say on the cover of the report here?” and he answers “Top Secret”.

    video:
    Return To Paradise - CBC Player

    It is noteworthy this piece mentions four dying in Chiang Mai, but they’re only talking about the four who were staying in the Downtown Inn (since demolished to, presumably, destroy evidence of aluminum phosphide). But there were two others who died in Chiang Mai around the same time, and at least one of whom had visited the Downtown Inn.

    And below is another video by CBC Canada on a woman who brought some aluminum phosphide pellets back from Pakistan and put them in her apartment with four children present. Of note is the toxicologist interviewed. He gives you a really good idea of how dangerous this stuff is.

    video:
    Pesticide used by Fort McMurray mother caused baby's death - CBC Player

    The odor given off is often like rotting fish or garlic, but it doesn’t always have any odor.

    Pediatr Emerg Care. 2011 Sep;27(9):869-71. doi: 10.1097/PEC.0b013e3182348e40.
    Unintentional fatal phosphine gas poisoning of a family. Lemoine TJ1, Schoolman K, Jackman G, Vernon DD.
    Author information 1Division of Pediatric Critical Care Medicine, Department of Pediatrics, University of Utah, Salt Lake City, UT, USA.

    A family of 6 members was exposed to phosphine gas after their yard was treated with aluminum phosphide for a rodent infestation. The inhalation of phosphine gas initially caused symptoms of abdominal distress in all of the family members. Within 36 hours of exposure, the 4-year-old sibling died of cardiopulmonary failure in a local emergency department. After an initial presentation of respiratory distress, the 15-month-old toddler progressed to complete cardiopulmonary collapse and did not survive despite aggressive intervention including extracorporeal membrane life support.
    CONCLUSIONS: Unintentional phosphine gas exposure is rare but has a toxic profile that results in a high fatality rate with no known antidote. (end)

    I remember this story. It was the YARD of the Utah family where the pellets were placed . The pellets were put in the ground by a licensed professionals, in the tunnels of gophers (I think) and it was the fumes carried through the tunnels in the yard up to close to the house, where the fumes found their way inside.

    In India, eating—not breathing—aluminum phosphide is a common method of suicide.

    'If you’re asleep in the room next to it or in the room where it’s been under fumigation, you’ll be dead in a few hours' - Denis Bureau, fumigation expert, on aluminum phosphide Deaths of Quebec women in Thailand may have been caused by pesticide - CBC News - Latest Canada, World, Entertainment and Business News
    Last edited by boi; 10-09-2016 at 06:57 PM.

  22. #672
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    Great detective work by the TD sleuthes , boi gives you credit for cracking this case.

  23. #673
    Thailand Expat
    Bower's Avatar
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    Sep 2009
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    15-10-2020 @ 05:33 PM
    Location
    South coast UK
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    Well done guys.

  24. #674
    Thailand Expat

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    Sep 2014
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    Quote Originally Posted by taxexile View Post

    I am in the uk now, and missing thailand intensely. The easygoing chaos, the friendliness of most of the social exchanges and the sheer attractiveness of the people.

    The ugliness and obesity that surrounds me on the streets here is nauseating, the streets of the town centre are awash with fat freaks,strange looking youth with facial piercings and frightening stares, and worst of all, decrepit poverty stricken pensioners with crooked backs and thirty year old raincoats, lined puffy faces and gloomy sunken eyes, hobbling into and out of the pound stores carrying ripped plastic shopping bags, admittedly I am in an ugly northern town, and the leafy suburbs where I am billeted are quite pleasant, but as soon as my obligations here have been undertaken I shall head off for the countryside, and no country on earth can beat the english at countryside.
    Trawling through this old thread I came across this little gem from Tax. It seems he has since had some sort of epiphany and is now content to be just another old codger among many. A victim of those decrepit northern body snatchers no doubt.

  25. #675
    Thailand Expat

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    25-03-2021 @ 08:47 AM
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    36,437
    You spelled trolling incorrectly, old bean...You're welcome...

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